My training was so specific and specialized, that I was able to go into the gym, knock out 4 workouts per week and never injure myself. I thought I was fucking bulletproof. I think my narrow specialty allowed for me to never have to experience burnout or trauma.
Maybe I should have quoted this in my original reply:
I have quite often felt this since my injuries (or as I said, succumbing to my injuries, since I do not believe it was a single injury, and more likely the pain just eclipsed my ability to resist that pain.
155x8,8,7 <-- RPE 9.0, very nice and super strong/stable
3D SMITH JM PRESS
BO BB ROWS
very nice and strong, except my last set LOL
I began the war on my hip flexor yesterday. I've been doing glute activation drills and postural work before that, but something felt off. I took inventory and realized I haven't been able to maximally contract my right glute in maybe 2 years now. Thought about it, and of course the hip flexor does antagonize the glute. Did some reading that suggested many of the things I was thinking about doing. Anyways, started on some stretches and I'm feeling some relief already. OHP felt so good today, and I could feel my glutes getting very sore during bo rows. So I think it's working. My right shoulder also felt stronger and more stable during both OHP and rows. Hope I'm onto something.
Yeah, that's why I drew the comparison.
Might be interesting to add it in again now that I'm better, diet-wise. I think all of the things I tried for so many years were for naught, because you can only do so much when you're in a 1-2000 kcal surplus daily.
Biomed Pharmacother 2020 Jul;127:110137. doi: 10.1016/j.biopha.2020.110137. Epub 2020 Apr 27.
The effect of Berberine on weight loss in order to prevent obesity: A systematic review
Zahra Ilyas 1, Simone Perna 1, Salwa Al-Thawadi 1, Tariq A Alalwan 1, Antonella Riva 2, Giovanna Petrangolini 2, Clara Gasparri 3, Vittoria Infantino 4, Gabriella Peroni 5, Mariangela Rondanelli 6
This study provides a critical overview of experimental studies in vitro, in humans, and in animals that evaluated the efficacy of Berberine and its effect on management of obesity and the related metabolic consequences. As a result of this review, we summarized the effects of Berberine in different models and the related mechanism of actions. In preclinical models, Berberine demonstrates that it affects gut microbiota by reducing diversity of microbes starting at a dosage of 100 mg/kg/day. Moreover, in animal models, Berberine explicates an action on glucose through the inhibition of α-glycosidase at a dose of 200 mh/kg/day. Berberine is also known to be effective against differentiation of adipocytes through a decrease in LXRs, PPARs, and SREBPs expression at 150 mg/kg/day. Other mechanism ascribed to Berberine are related to its inhibition of hepatic gluconeogenesis through the Phospheoenolpyruvate carboxykinase (PEPCK), Glucose-6-phosphate (G6Pase) and AMP-activated protein kinase (AMPK). Furthermore, Berberine (associated to Red Yeast Rice) is effective in decreasing lipid levels in rats, which consequently lowers the change of weight gain at dosage of 40 mg/kg to 380 mg/kg/day. All the above preclinical data are confirmed in human studies where Berberine can modulate the diversity of gut microbes at the dose of 500 mg/day. In addition, Berberine is found to have a beneficial impact on gene regulation for the absorption of cholesterol at a daily dose of 300 mg in humans, an amelioration on glucose accumulation at 1.0 g daily dose was also observed. For all these reasons, this review gives an important good account of the impact of Berberine in obesity treatment and prevention.